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	<title>HealthPDF.org</title>
	<link>http://www.healthpdf.org/</link>
	<description><![CDATA[Download health related pdf and ebook for free. browse well categorized library or use search bar to locate the item you are needed]]></description>
	<pubDate>Thu, 16 Aug 2007 15:18:33 -0400</pubDate>
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		<title>Mold: Causes, Health Effects And Clean-Up</title>
		<link>http://www.healthpdf.org/allergy/mold-spores/mold-causes-health-effects-and-clean-up.html</link>
		<pubDate>Sat, 20 Dec 2008 00:03:23 -0500</pubDate>
		<dc:creator>admin</dc:creator>
	<category>Mold Spores</category>
		<guid isPermaLink="false">http://www.healthpdf.org/allergy/mold-spores/mold-causes-health-effects-and-clean-up.html</guid>
		<description><![CDATA[Joseph Lstiburek, Ph.D., P.Eng. Nathan Yost, MD Terry Brennan, MS Mold requires water. No water, no mold. Mold is the result of a water problem. Fix the water problem. Clean up the mold. And you have fixed the mold problem. ...]]></description>
			<content:encoded><![CDATA[Mold: Causes, Health Effects and Clean-Up Joseph Lstiburek, Ph.D., P.Eng. Nathan Yost, MD Terry Brennan, MS Mold requires water. No water, no mold. Mold is the result of a water problem. Fix the water problem. Clean up the mold. And you have fixed the mold problem. To avoid mold problems, avoid water problems. Design and build in a manner that reduces water problems. Mold also requires food. The food it likes best is cellulose the more processed the better. Mold really likes wet paper. It kind of likes wet wood, but not as much as it likes wet paper. It likes processed wood better than it likes real wood. So mold likes oriented strand board (OSB) better than plywood and plywood better than a stud or a joist. Mold also likes the feces of cockroaches and dust mites, as well as some pastes, paints and adhesives. Just because something gets wet, it doesnt mean it will get moldy. It needs to be wet for a while. Wet paper needs to be wet for a couple of days. Wet wood, for a couple of weeks. And it also needs to be warm. Warm, wet paper that is wet for a while is a problem. Because it usually takes time for mold to grow, promptly drying the building after a water event will prevent a mold problem from developing. Of course, make certain that the underlying problem that caused the water problem is also corrected. There are always going to be water problems. But if you fix the water problems quickly enough you wont have mold problems. You want to be able to see a water problem right away if you have one so that you can fix it right away. Water problems that you cant see for a long time are the type of problems that lead to bad mold problems. For example, wet paper faced gypsum board that you cant see is a problem. Like from a small plumbing leak. Especially in an exterior insulated wall. Why an insulated wall? The insula tion reduces the ability of the water to drain and leak out so that you can see it. Also, the insulation r ...]]></content:encoded>
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		<title>M05.8 Other Seropositive Rheumatoid Arthritis</title>
		<link>http://www.healthpdf.org/bone/dorsopathies/m05-8-other-seropositive-rheumatoid-arthritis.html</link>
		<pubDate>Fri, 19 Dec 2008 00:02:55 -0500</pubDate>
		<dc:creator>admin</dc:creator>
	<category>Dorsopathies</category>
		<guid isPermaLink="false">http://www.healthpdf.org/bone/dorsopathies/m05-8-other-seropositive-rheumatoid-arthritis.html</guid>
		<description><![CDATA[Other deforming dorsopathies. See site code, page 257. Excludes: ... Other dorsopathies, not elsewhere. classified. See site code, page 257. M53.0 ...]]></description>
			<content:encoded><![CDATA[M05.8 Other seropositive rheumatoid arthritis M05.8 Other seropositive rheumatoid arthritis  0-9  M05.9 Seropositive rheumatoid arthritis, unspecified  0-9  M06 Other rheumatoid arthritis See site code,  M06.0 Seronegative rheumatoid arthritis  0-9  M06.1 Adult-onset Stills disease  0-9  Excludes: Stills disease NOS (M08.2 -) M06.2 Rheumatoid bursitis  0-9  M06.3 Rheumatoid nodule  0-9  M06.4 Inflammatory polyarthropathy  0-9  Excludes: polyarthritis NOS (M13.0) M06.8 Other specified rheumatoid arthritis  0-9  M06.9 Rheumatoid arthritis, unspecified  0-9  M07* Psoriatic and enteropathic arthropathies See site code,  Excludes: juvenile psoriatic and enteropathic arthropathies (M09. -*) M07.0* Distal interphalangeal psoriatic arthropathy (L40.5)  0,4,7,9  M07.1* Arthritis mutilans (L40.5)  0-9  M07.2* Psoriatic spondylitis (L40.5) M07.3* Other psoriatic arthropathies (L40.5)  0-9  M07.4* Arthropathy in Crohns disease  regional enteritis  (K50.-)  0-9  M07.5* Arthropathy in ulcerative colitis (K51.-)  0-9  M07.6* Other enteropathic arthropathies  0-9  Replaces right column on  M08 Juvenile arthritis 1352 See site code,  Includes: arthritis in children, with onset before 16th birthday and lasting longer than 3 months Excludes: Feltys syndrome (M05.0 -) juvenile dermatomyositis (M33.0) M08.0 Juvenile rheumatoid arthritis  0-9  Juvenile rheumatoid arthritis with or without rheumatoid factor M08.1 Juvenile ankylosing spondylitis  0-9  Excludes: ankylosing spondylitis in adults (M45.0 -) M08.2 Juvenile arthritis with systemic onset  0-9  Stills disease NOS Excludes: adult -onset Stills disease (M06.1-) M08.3 Juvenile polyarthritis (seronegative) Chronic juvenile polyarthritis M08.4 Pauciarticular juvenile arthritis  0-9  M08.8 Other juvenile arthritis  0-9  M08.9 Juvenile arthritis, unspecified  0-9  M09* Juvenile arthritis in diseases classified elsewhere See site code,  Excludes: arthropathy in Whipples disease (M14.8*) M09.0* Juvenile arthritis in psoriasis (L40.5) ...]]></content:encoded>
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		<title>S O F T T I S S U E</title>
		<link>http://www.healthpdf.org/bone/soft-tissue/s-o-f-t-t-i-s-s-u-e.html</link>
		<pubDate>Fri, 19 Dec 2008 00:02:55 -0500</pubDate>
		<dc:creator>admin</dc:creator>
	<category>Soft Tissue</category>
		<guid isPermaLink="false">http://www.healthpdf.org/bone/soft-tissue/s-o-f-t-t-i-s-s-u-e.html</guid>
		<description><![CDATA[massage therapists feel a client's tissues and know the health of the tissue, ... Therapist And Client Experiences Of Soft Tissue ...]]></description>
			<content:encoded><![CDATA[SOFT TISSUE RESISTANCE BARRIERS MASTER BOATBUILDERS TOUCH A PIECE OF WOOD and know the type of tree it came from, what its strengths and weaknesses are, and where it will do the most good in the boat they are building. In a similar way, masterful massage therapists feel a clients tissues and know the health of the tissue, how the client experiences the therapists touch, and what types of massage techniques the client both needs and enjoys. This delightful experience (for both therapist and client) flows from the therapists sensitivity and experience. She has given a lot of treatments, and listened with her fingers to her clients flesh, and with her ears to the T H E S E T O O L S F O R T E A C H I N G , P R A C T I C I N G A N D C O M M U N I C A T I N G W I T H Y O U R C L I E N T S W I L L H E L P Y O U B E C O M E M O R E A T T U N E D T O T H E I R N E E D S . Ebby May/Stone S p r i n g 2 0 0 3 WWW.AMTAMASSAGE.ORG 99 Technique Compression is a key feature of many massage manipulations. Compression ranges from static compression with a thumb, finger, elbow or the heel of the hand, to petrissage manipulations, which have a significant component of compression to them. In this article, we are examining only the simplest scenario, which is static compression. Take a moment and use your dominant hand to feel the tissues of the nondominant arm. Gently squeeze your forearm between thumb and fingers in several locations, noticing how the different locations in the forearm give very different resistance to the compressive force of the palpating hand. It is through this quality of resistance that we know we are contacting bone, perhaps a thin layer of muscle over bone, or a complex tangle of ropes that are the forearm muscles and their tendons! Now that you have had an initial feel of your forearm, lets use it to explore resistance barriers to compression. Find a band of muscle that feels harder, ...]]></content:encoded>
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		<title>Genital Warts</title>
		<link>http://www.healthpdf.org/skin/warts/genital-warts.html</link>
		<pubDate>Fri, 19 Dec 2008 00:02:55 -0500</pubDate>
		<dc:creator>admin</dc:creator>
	<category>Warts</category>
		<guid isPermaLink="false">http://www.healthpdf.org/skin/warts/genital-warts.html</guid>
		<description><![CDATA[Genital warts are caused by the Human Papillomavirus or HPV. Genital HPV is the most common sexually transmitted infection in the United States. About 24 million people are currently infected with genital HPV. ...]]></description>
			<content:encoded><![CDATA[Genital warts are caused by the Human Papillomavirus or HPV. Genital HPV is the most common sexually transmitted infection in the United States. About 24 million people are currently infected with genital HPV. Up to 80% of men and women will become infected at some time in their life. WHAT IS HPV? HPV, like all viruses, is a very small organism that needs to infect cells in order to survive. There are more than 120 known types of HPV. Some types cause warts on the hands or feet. Other types cause genital warts or condyloma. There are also types of HPV that infect the cervix (lower part of the uterus) where they can cause either harmless cell changes or potentially precancerous cell changes or dysplasia. (See Human Papillomavirus handout). A person can be infected simultaneously with multiple HPV types. Most people who become infected with genital HPV will not have any symptoms and will not know they have an infection. It has been estimated that fewer than 5% of people with genital HPV will develop warts. The others have subclinical (or invisible) HPV. Whether people with subclinical HPV infection are as contagious as people who have visible warts is not known. HOW DO YOU GET HPV AND WHEN DO WARTS APPEAR? HPV infection is usually spread by direct skin-to-skin contact during vaginal, oral, or anal sex with an infected individual. After exposure, warts may take weeks, months or even years to appear; or they may never appear at all. For this reason, ...]]></content:encoded>
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		<title>Deflazacort For Type-1 Autoimmune Hepatitis In A Korean Girl</title>
		<link>http://www.healthpdf.org/liver/autoimmune-hepatitis/deflazacort-for-type-1-autoimmune-hepatitis-in-a-korean-girl.html</link>
		<pubDate>Thu, 18 Dec 2008 00:05:38 -0500</pubDate>
		<dc:creator>admin</dc:creator>
	<category>Autoimmune Hepatitis</category>
		<guid isPermaLink="false">http://www.healthpdf.org/liver/autoimmune-hepatitis/deflazacort-for-type-1-autoimmune-hepatitis-in-a-korean-girl.html</guid>
		<description><![CDATA[Autoimmune hepatitis is a chronic necro-inflammatory hepatitis of unknown etiology. It is characterized by autoantibodies that target against liver-specific and non-organspecific antigens ...]]></description>
			<content:encoded><![CDATA[Autoimmune hepatitis is a chronic necro-inflammatory hepatitis of unknown etiology. It is characterized by autoantibodies that target against liver-specific and non-organspecific antigens (1). Corticosteroids, especially prednisone or prednisolone (PD), have been the key drugs of initial antiinflammatory and immuno-suppressive therapy of autoimmune hepatitis in children (2). However, it is well known that long-term use of corticosteroid is associated with the risk of steroid-induced toxicities, and this requires newer immunosuppressive agents for the treatment of autoimmune hepatitis, especially in growing children. We report on the case of a 11-yr-old Korean girl with type1 autoimmune hepatitis who discontinued PD due to steroidinduced toxicities, but who remained in biochemical and clinical remission while undergoing deflazacort and ursodeoxycholic acid (UDCA) combination therapy. CASE REPORT A 9-yr-old girl was admitted to Eul-Ji Hospital because of jaundice and right upper quadrant pain. Her height was 135 cm (75-90 percentile), and weight was 43 kg (over 97 percentile). The sclera was icteric, and the liver edge descended 4 cm below the right costal margin. The results of laboratory studies were as follows: aspartate aminotransferanse (AST) 1,106 IU/L, alanine aminotransferase (ALT) 1,888 IU/L, total bilirubin 25.7 mg/dL, direct bilirubin 18.2 mg/dL, total protein 6.9 g/dL, albumin 4.2 g/dL, serum alkaline phosphatase 1,160 IU/L, GGTP 71 IU/L, INR 2.4, aPTT 120 sec, antinuclear antibody (ANA) (+) speckled pattern, with 1:320 titer, anti-smooth muscle antibody (Ab) (-), antimitochondrial (AMA Ab) (-), IgG 1,397 mg/dL, anti-HAV IgM (-), anti-HBc IgM (-), HbsAg (-), EBV-VCA IgG/M (-/-), and ceruloplasmin 28 mg/dL. Liver biopsy results showed piecemeal necrosis at the portal-lobular interface accompaning mild to moderate infiltration of lymphocytes, moderate centrilobular cholestasis, ...]]></content:encoded>
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		<title>What Is Phenylketonuria (PKU)?</title>
		<link>http://www.healthpdf.org/genetic/phenylketonuria/what-is-phenylketonuria-pku.html</link>
		<pubDate>Thu, 18 Dec 2008 00:05:38 -0500</pubDate>
		<dc:creator>admin</dc:creator>
	<category>Phenylketonuria</category>
		<guid isPermaLink="false">http://www.healthpdf.org/genetic/phenylketonuria/what-is-phenylketonuria-pku.html</guid>
		<description><![CDATA[PKU is an inherited disorder in which the baby is unable to use a certain part ofprotein (phenylalanine) found in food and milk. PKU occurs in about 1 in 8,800 Michigan newborns. ...]]></description>
			<content:encoded><![CDATA[PKU is an inherited disorder in which the baby is unable to use a certain part ofprotein (phenylalanine) found in food and milk. PKU occurs in about 1 in 8,800 Michigan newborns. Without treatment, phenylalanine will build up in a baby&acirc; sbody and mental retardation will result. br /br /How may PKU affect my child?br /br /Developmental Delay andMental RetardationMental retardation will occur if a childwith PKU does not begin a specialdiet within the first 7 to 10 days oflife. If the special diet is started at thecorrect time and followed properly,a child should not have mentalretardation due to PKU.  ...]]></content:encoded>
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		<title>USVH Disease Of The Week  4: Schizophrenia</title>
		<link>http://www.healthpdf.org/brain/schizophrenia/usvh-disease-of-the-week-4-schizophrenia.html</link>
		<pubDate>Thu, 18 Dec 2008 00:05:38 -0500</pubDate>
		<dc:creator>admin</dc:creator>
	<category>Schizophrenia</category>
		<guid isPermaLink="false">http://www.healthpdf.org/brain/schizophrenia/usvh-disease-of-the-week-4-schizophrenia.html</guid>
		<description><![CDATA[Schizophrenia, among the most disabling severe mental disorders, affects 1 percent of the ... Schizophrenia is a syndrome characterized by disordered ...]]></description>
			<content:encoded><![CDATA[USVH Disease of the Week  4: Schizophrenia USVH Disease of the Week  4: Schizophrenia Schizophrenia in Older Adults Kansas State University Department on Aging / Published in 1999 Schizophrenia, among the most disabling severe mental disorders, affects 1 percent of the population. It is a severe, persistent mental disorder that requires long-term health care. It has a dramatic, debilitating effect on most aspects of everyday functioning, behavior, and personal experience, and it can have a devastating effect on families as well. Symptoms of schizophrenia begin to emerge when a person is in young adulthood. The long-term outcome for a person with schizophrenia varies. For older people who have experienced some relief from their symptoms with improved medications and who have strong family and/or social support systems, substantial improvement in their symptoms and functioning can occur. For older individuals who developed the disorder in their younger years, did not have the ...]]></content:encoded>
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		<title>Bipolar Disorder</title>
		<link>http://www.healthpdf.org/brain/bipolar-disorder/bipolar-disorder-2.html</link>
		<pubDate>Wed, 17 Dec 2008 00:07:49 -0500</pubDate>
		<dc:creator>admin</dc:creator>
	<category>Bipolar Disorder</category>
		<guid isPermaLink="false">http://www.healthpdf.org/brain/bipolar-disorder/bipolar-disorder-2.html</guid>
		<description><![CDATA[WHAT IS BIPOLAR DISORDER? Bipolar disorder, or manicdepressive illness, is characterized by moods that swing between two opposite poles: Periods of mania (with exaggerated euphoria, irritability, or both). ...]]></description>
			<content:encoded><![CDATA[WHAT IS BIPOLAR DISORDER? Bipolar disorder, or manicdepressive illness, is characterized by moods that swing between two opposite poles: Periods of mania (with exaggerated euphoria, irritability, or both). To qualify for a diagnosis of bipolar disorder, only one episode of mood elevation needs to be reported. Episodes of depression. Although chemical imbalances in the brain are a key component of bipolar disorder, it is a complex condition that involves genetic, environmental and other factors. Bipolar Disorder Categories Bipolar disorder is classified as bipolar disorder I, bipolar disorder II, or cyclothymic disorder according to the pattern and severity of the symptoms. These conditions are most likely part of a single spectrum of the disease; patients with one type may develop another. Nevertheless, they are distinct enough to merit separate classifications, and some experts believe these conditions are actually separate disorders with different biologic factors that account for their differences. Bipolar Disorder I. Bipolar disorder I is characterized by at least one manic episode, with or without major depression. In 60% to 70% of cases, manic episodes precede or follow depressive episodes in a regular pattern. Episodes are more acute and severe than in the other two categories. Without treatment, patients average four episodes of dysregulated mood each year. With mania, either euphoria or irritability may mark the phase. In addition, there are significant negative effects (such as sexual recklessness, excessive and impulsive shopping, and sudden traveling) on a patients social life, performance at work, or both. Untreated mania lasts at least a week and it can last for months. Typically, depressive episodes tend to last six to 12 months, if left untreated. Bipolar Disorder II and Hypomania. Bipolar disorder II is characterized by at least one episode of hypomania and at least one episode of major depression. With hypomania ...]]></content:encoded>
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		<title>EDICAL</title>
		<link>http://www.healthpdf.org/skin/skin-tags/edical.html</link>
		<pubDate>Wed, 17 Dec 2008 00:07:49 -0500</pubDate>
		<dc:creator>admin</dc:creator>
	<category>Skin Tags</category>
		<guid isPermaLink="false">http://www.healthpdf.org/skin/skin-tags/edical.html</guid>
		<description><![CDATA[Skin tag removal is considered medically necessary when there is documentation from the physician that the lesion has been subject to irritation/trauma resulting in inflammation, bleeding or infection. ...]]></description>
			<content:encoded><![CDATA[Skin tag removal is considered medically necessary when there is documentation from the physician that the lesion has been subject to irritation/trauma resulting in inflammation, bleeding or infection. br /br /  General Backgroundbr /br /  Skin tags, or acrochordons, are benign, soft, fleshy tumors that typically appear in adulthood (i.e., age 60 and over). They are found in 25% of the population and more common in women. The underlying cause is unknown, but may be hereditary. These skin lesions are flesh-colored or hyperpigmented, often pedunculated (i.e., attached to the skin by a thin stalk), and usually occur on the eyelids, neck, axillae or groin. br /br /  Skin tag lesions are composed of hyperplastic epidermis covering a dermal connective tissue stalk. They often appear in multiple numbers and may vary in size from one millimeter to one centimeter in diameter. Skin tags may be associated with seborrheic keratosis, a benign hyperkeratotic lesion of the epidermis. Lesions can increase in size and number with pregnancy or weight gain. It is not unusual for skin tags to return after removal. Due to the benign nature of skin tags, they rarely require pathologic examination. br /br /  In the majority of cases, skin tags are asymptomatic, benign and require no intervention. Patients often seek treatment because of the unsightly appearance of skin tags, requesting removal solely for cosmetic purposes. In some limited cases, skin tags may be subject to repeated local trauma or irritation resulting in chronic inflammation, pain, bleeding or localized infection. In these situations intervention may be medically necessary.br /br /  Medical treatment of skin tags includes avoidance of recurrent trauma or irritation (e.g., avoiding irritating jewelry or tight-fitting clothes) and the application of topical medications such as an antibiotic ointment. br /br /  Surgical treatment of skin tags includes excision with scissors, cautery or cryotherapy with liquid nitrogen. Skin tag removal is considered cosmetic in nature and not medically necessary when performed solely to improve appearance or to treat psychological symptomatology or psychosocial complaints. br /br /  Summarybr /br /  Skin tags, or acrochordons, are fleshy tumors composed of hyperplastic epidermis covering a dermal connective tissue stalk. Most are asymptomatic and benign requiring no intervention. If signs of irritation, inflammation, bleeding, and/or infection are persistent, medical or surgical intervention would be indicated.br /br /  -- download file to read the full content --]]></content:encoded>
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		<title>Asbestos: Health And Prevention</title>
		<link>http://www.healthpdf.org/lungs/asbestosis/asbestos-health-and-prevention.html</link>
		<pubDate>Wed, 17 Dec 2008 00:07:49 -0500</pubDate>
		<dc:creator>admin</dc:creator>
	<category>Asbestosis</category>
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		<description><![CDATA[fibers, may increase risk of asbestosis. fibers, may increase risk of asbestosis ... asbestosis? asbestosis? Treatment involves treating symptoms and ...]]></description>
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